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The rupture of the physical barrier (OMM) that limits their release into the cytosol constitutes a point-of-no-return in cell death ( c release, whereas Bcl2-family death antagonists prevent it

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However, at the end of the stress episode, ROS concentration need to return to normal level to avoid HSCs exhaustion

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We evaluated adipogenic and angiogenic activities by immunofluorescence and observed a higher number of intact perilipin-positive mature adipocytes in the GSH-treated group than in the control group