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These toxins can lead to subtle cellular changes such as interfering with the methylation of DNA (Rooney 2011)

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Like so many other aspects of the condition, the reason the GI tracts inflammatory response becomes imbalanced in individuals with IBS is not fully understood

Via this metabolic adaptation, cells can continually generate TCA metabolites, such as -KG and citrate, which are converted to cytosolic acetyl-CoA for lipid biosynthesis (Fig

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