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Thus, these comorbid conditions may further exacerbate the proinflammatory endothelial and monocyte phenotypes associated with preexisting G6PD-d, thereby compounding CVD risk 28,29,30

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The A1298C variant causes less of a change to the way the enzyme works, with two copies of the variant decreasing enzyme function by around 20%

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To assess the possible role of SA-mediated and GSH-associated defense gene expression in antiviral responses at this advanced stage of TMV pathogenesis we monitored transcript induction of two pathogenesis-related and two GST genes ( NtPR-1a , NtPRB-1b, NtGSTtau, NtGSTphi )