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Inadequate sleep blunts GH response limiting tissue repair and peptide effectiveness

10.1016/j.cell.2011.02.013 2

Hence, elevated oxidative stress in the cornea is likely not a result of aging or cumulative DNA damage [71] but rather thought to be partly attributed to acquired defects in mitochondrial function or ROS-scavenging abilities, leading to broad cellular stress in the corneal stroma, decreased collagen expression, keratocyte loss, and, ultimately, corneal thinning [72]

JCI Insight 5 , e140532 (2020)