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high-quality RCT evidence specifically supporting these formulations for joint pain outcomes is limited

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Differential pathophysiological mechanisms between CHS and EHS While CHS and EHS share core pathogenic pathways (e.g., hyperthermia, inflammation, cell death), their mechanistic features differ significantly due to distinct patient demographics and triggers: Inflammatory kinetics EHS (predominant in young, active individuals) is characterized by rapid, exertion-amplified inflammatory responses

PMCID: PMC3533412

The same investigation was conducted with benzyl benzoate and water