This mechanism leads to decreased expression of hypoxia-inducible factor (HIF)-1 and Tf while boosting the levels of proteins such as FTH1, NAD(P)H quinone dehydrogenase 1 (NQO1), HO-1, and GPX4
& Miyadera, T
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Mitochondrial reactive oxygen species enhance AMP-activated protein kinase activation in the endothelium of patients with coronary artery disease and diabetes
not intended as therapeutic claims
