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Masman et al

doi: 10.1126/science.289.5482.1159

5.2.2 Mitochondrial dysfunction Ketamine disrupts mitochondrial function by reducing the membrane potential, increasing cytochrome c release, and promoting ROS generation, which collectively induce neuronal apoptosis ( Human studies have identified metabolic abnormalities in ketamine abusers, particularly in the prefrontal cortex, which may reflect mitochondrial dysfunction (Liao et al., 2011)

13 Studies investigating the status of claudins and other TJ-associated proteins in human biopsy specimen from chronic inflammatory conditions (Inflammatory Bowel Disease (IBD)) 14 have demonstrated differential changes in their total cellular content and/or distribution (see Table 1)