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could account for developmental and neurochemical psychopathology observed after nicotine exposure[74] That is, nicotine may incorrectly mimic or duplicate endogenous signals produced by acetylcholine ACh, which an important part when synaptic formation is normal, by functioning as a nAChR agonist [78]

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Some level of e-cigarette dependence was found among 86.8% (n=514) of participants, and 73.5% had tried to quit using e-cigarettes

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