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For esophageal repair, stronger collagen architecture provides structural support for regenerating epithelium and contributes to overall tissue resilience against future injury

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During ischemia, dying neurons release massive amounts of glutamate, which overstimulates NMDA receptors on neighboring neurons, causing calcium influx and further cell death

This protection becomes especially important during GHK-Cu protocols when new collagen is being actively synthesized and would be vulnerable to UV damage