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Under basal homeostatic conditions, Nrf2 is sequestered in the cytoplasm through its interaction with the adaptor protein KEAP1 (Kelch-like ECH-associated protein 1), which targets Nrf2 for ubiquitination and subsequent proteasomal degradation

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This ultimately promotes abnormal cell proliferation, inflammation, angiogenesis, immune escape and tissue infiltration

Immune checkpoint inhibitors that target this pathway have shown promise as a treatment for metastatic cervical cancer

This symbiotic association played a crucial role in improving the uptake of essential nutrients such as potassium, calcium, and magnesium under salt-stress conditions