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Antigen-specific cellular hyporesponsiveness in a chronic human helminth infection is mediated by TH3/TR1-type cytokines IL-10 and transforming growth factor- but not by a TH1 to TH2 shift

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It is now considered the most abundant isozyme of the GPx family and is present ubiquitously in all the tissues of human origin [1, 8, 9]

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