Oxidative stress activates protein tyrosine kinase and matrix metalloproteinases leading to blood-brain barrier dysfunction
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This review outlines the metabolic transitions observed during AKI, emphasizing the underlying pathophysiological mechanisms and key regulatory pathways implicated in mitochondrial dysfunction and energy metabolism
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10.1371/journal.pone.0085933 14 Bodner-AdlerB.BodnerK.KimbergerO.CzerwenkaK.LeodolterS.MayerhoferK
