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revealed that CKD patients had poorer clinical cardiovascular outcomes in association with enhanced inflammation and oxidative stress, as evidenced by their elevated MDA-modified LDL levels and peak serum C-reactive protein (CRP) [43]

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Also, hypomethylation of GRP78, FTO and TNF and PDGFR-, and hypermethylation of leptin, PPARG, LXR, Lipin 1 and GRP78 in mothers have been shown to predispose offspring to obesity, disruption of hepatic lipid metabolism and other cardiometabolic outcomes [164]

This pathway has been shown to contribute to synaptic dysfunction, neuroinflammation and glutamate dysregulation 60