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isotonic with the endolymph and/or perilymph) upon delivery at the target site

Meanwhile, hyperglycemia produces AGEs (advanced glycation end products), PKC (protein kinase C), free fatty acids, and cytokines, thereby activating the NADPH oxidation system in renal cells [54, 55]

The significantly increased rate of hyperlipidemia in the hernia group is most likely related to the increased incidence of obesity associated with HHs

CPT-II on the inner membrane regenerates fatty acyl-CoA for beta-oxidation, releasing free carnitine to recycle through the shuttle